Finding into death of GM
A 19-year-old female with anorexia nervosa, borderline personality disorder, and chronic suicidality was admitted to St Vincent’s psychiatric inpatient unit on 28 July 2023. She reported ongoing suicidal thoughts but eng…
Deceased
Paul Bernard Finck
Demographics
47y, male
Coroner
Coroner Audrey Jamieson
Date of death
2007-09-19
Finding date
2015-02-26
Cause of death
pulmonary thromboembolism and deep vein thrombosis
AI-generated summary
Paul Finck, 47, an involuntary psychiatric patient with schizophrenia, died from pulmonary thromboembolism and deep vein thrombosis while admitted to the psychiatric ward. He presented with fever, tachycardia, elevated CK levels and dehydration from walking 26km barefoot. Clinical staff considered differential diagnoses including neuroleptic malignant syndrome and infection. Despite family concerns about immobilization from sedation potentially contributing to DVT formation, the coroner found his presentation was not diagnostic of DVT, clinical staff did not predict this complication, and appropriate monitoring was provided. The condition was found unforseeable and unlikely even with admission to a medical ward. No adverse findings were made against healthcare staff.
AI-generated summary — refer to original finding for legal purposes. Report an inaccuracy.
Specialties
Drugs involved
Court Reference: 2007 3703
Form 37 Rule 60(1) Section 67 of the Coroners Act 2008
Inquest into the Death of: PAUL BERNARD FINCK Delivered On: 26 February 2015
Delivered At: Coroners Court of Victoria 65 Kavanagh Street, Southbank
Hearing Dates: 26 and 27 October 2011 Findings of: AUDREY JAMIESON Representation: Ms D Foy of Counsel on behalf of Eastern Health
Police Coronial Support Unit Leading Senior Constable Greig McFarlane
I, AUDREY JAMIESON, Coroner, having investigated the death of PAUL BERNARD FINCK
AND having held an inquest in relation to this death on 26 October 2011 at MELBOURNE
find that the identity of the deceased was PAUL BERNARD FINCK born on | September 1960
and the death occurred on 19 September 2007
at Maroondah Hospital, Davey Drive, Ringwood East, 3135
from:
l(a) PULMONARY THROMBOEMBOLISM 1(b) DEEP VEIN THROMBOSIS
in the following circumstances:
On 26 October 2011, a mandatory inquest under section 52(2)(b) of the Coroners Act 2008 (Vic)! began into the death of Mr Paul Bernard Finck, because immediately before his death, Mr Finck was “a person placed in....care” as it is defined in the Act, in that prior to his death, Mr Finck was a patient in an approved mental health service within
the meaning of the Mental Health Act 1986 (Vic) (the Mental Health Act).?
On 15 September 2007, Mr Finck was admitted as an involuntary patient to the Psychiatric unit of Maroondah Hospital after walking barefoot to his Aunt’s home some considerable distance away. Whilst in the Psychiatric unit, Mr Finck was assessed and was placed under observation. On 19 September 2007, at approximately 7.45pm, staff found Mr Finck collapsed in his ensuite. Cardiopulmonary resuscitation (CPR) was
performed, however, Mr Finck was unable to be revived.
Mr Paul Finck, 47 years of age at the time of his death, resided in Nunawading with two
housemates and was in receipt of a Disability Support Pension.
Mr Finck had a history of mental health issues after being diagnosed with Schizophrenia in his late teens/early twenties. He had previously been admitted to the Eastern Health
Mental Health Service in October/November 2002 following separation from his then
' See below discussion under the heading of “Jurisdiction” for further explanation.
2 As it then was.
wife, Since 2002, he had been managed by his General Practitioner (GP) and private Psychiatrist. Tt was noted that he had good support from family and community
workers.
On 14 September 2007, Mr Finck arrived at his Aunt’s house in Mulgrave. It is believed that he had walked all the way from his house in Nunawading. Mr Finck stated that he had walked all night. He was barefoot and said that he had lost his shoes and
consequently received injuries to the balls of his feet.
He was described as disorientated upon his arrival. His Aunt rang his mother, who came
and collected him.
Mr Finck was taken by his mother to the Emergency Department (ED) at Maroondah Hospital at 12.37pm on 15 September 2007. Dr Ambreen Memon consulted with him and noted that he had missed his medication (Abilify/Aripiprazole) over the last three days. She noted that he had walked up to 26km as he was feeling upset. She also noted
that he looked confused, had a vacant stare and his speech was slow with a blunt affect.
Dressings were applied to his feet and a referral was made for Psychiatric assessment by a Registered Psychiatric Nurse. Mr Finck was then admitted to Maroondah Hospital, West Ward on 15 September 2007 as an involuntary Psychiatric patient for Schizophrenia and psychotic episodes. On admission, he was found to be febrile and tachycardic. Abnormalities were seen in his Thyroid Function Tests and he was possibly
experiencing urinary retention.
Mr Finck’s brother reported him to be in a Catatonic state initially upon admission.’
Hospital staff found his lack of engagement a barrier to assessing his psychotic symptoms."
Mr Finck received both medical and Psychiatric care whilst in the West Ward’s High Dependency Unit (HDU). He was referred to the Endocrinology unit, and a range of clinical tests were ordered including an ECG, blood cultures, urine samples, nuclear thyroid scan, chest X-ray and a bladder scan. He was commenced on oral antibiotics on
17 September 2007.
3 Transcript p. 9
“ Statement of Psychiatrist Dr Ashu Gandhi, undated, coronial brief of evidence p. 16.
il.
On 18 September 2007, Consultant Psychiatrist Dr Ashu Gandhi advised that Mr Finck’s antipsychotic medication should be ceased,° that his vital signs should be regularly monitored, and that he should be transferred to the ED if his condition deteriorated.® Mr Finck was also reviewed by Endocrinologist Resident Dr Xiang Loh in the morning, who determined the differential diagnoses to include infection, thyrotoxicity,
hyperthyroidism, urinary retention and neuroleptic malignant syndrome (NMS).’
In the afternoon of 18 September 2007, Mr Finck was reviewed by the Medical Registrar who considered it was likely he was suffering from a urinary tract infection causing retention and fever, with the rise in Creatine Kinase (CK) noted in his blood resulted attributed to walking 26kms. It was thought that it was unlikely that he was suffering
from NMS
On 19 September 2007, Mr Finck was reviewed by Dr Gandhi in the morning and was reported to have passed urine, with stable vital signs, and was observed to be generally settling down,’ with mildly increased engagement, although still in an apparent catatonic
state.
At approximately 11.30am on Wednesday 19 September 2007, Mr Finck was transferred from the HDU to the Low Dependency Unit (LDU). Mr Finck also complained of cpigastric pain later that day. As a result, Psychiatric Registrar Dr David Harms requested Mr Finck to be reviewed by the Psychiatric Registrar, Dr Piyumali de Silva,
that evening,
At approximately 7.30pm, Dr de Silva walked to the West Ward nurses’ station. On her way, she observed Mr Finck in his room sitting upright in a lounge chair facing the corridor.
Dr de Silva then spent some time reading his history and speaking with the Nursing staff
about Mr Finck and other patients on the ward. Nurse Fiona Lankford alerted Dr de
Due to the possibility of Mr Finck having neuroleptic malignant syndrome; transcript p. 62 and 135.
5 Statement of Dr Ashu Gandhi, undated, coronial bricf of evidence p. 16.
7 Statement of Dr Xiang Ho dated 26 October 2010, coronial brief of evidence p.18E.
® Statement of Medical Director of Maroondah Hospital Dr Peter Sloan dated 24 August 2010, coronial brief of evidence, p. 18G.
° Statement of Dr Ashu Gandhi, undated, coronial brief of evidence p. 16, although Dr Gandhi notes that a record of this was not located.
19,
Silva to the abdominal pain as well as mild tachycardia and hypertension that she had
observed just before Dr de Silva’s arrival.
At approximately 7.43pm, Dr de Silva and Nurse Lankford entered Mr Finck’s room.
He was not in his bedroom and was located in the ensuite, collapsed on the ground
between the toilet and the wall.
Dr de Silva attended to him, and she thought that she detected a weak pulse that stopped almost immediately. Nurse Lankford called a ‘code blue’, with the code blue response team arriving within two minutes. Prior to their arrival, Dr de Silva removed Mr Finck from the wedged position that she located him in, cleared debris from his mouth, applied
oxygen and commenced manual ventilation.
Once the code blue response team arrived, full resuscitation efforts were employed including intubation, defibrillation and the administration of adrenaline. However, they
were unable to restore spontaneous circulation and the decision was made to cease
resuscitation efforts at 8.14pm, upon which time Mr Finck was formally pronounced
deceased.
Identity
The identity of Paul Bernard Finck was without dispute and required no further
investigation,
The medical investigation
21,
On 25 September 2007, Forensic Pathologist Dr Sarah Parsons from the Victorian Institute of Forensic Medicine performed an autopsy on the body of Mr Finck, reviewed a post mortem CT scan and the Victoria Police Report of Death, Form 83. Anatomical findings included one abrasion on the right upper forearm and another abrasion overlying the right portion of the face, between the right eye and right ear. Dr Parsons also indentified deep venous thromboses in the left and right calves and a large saddle pulmonary embolism in Mr Finck’s lungs. Dr Parsons noted that Mr Finck had been running a fever in the days prior to his death and was dehydrated, which she opined may
have increased his risk of pulmonary thromboembolism.
Toxicological analysis of Mr Finck’s blood revealed the presence of diazepam, its
metabolite nordiazepam and olanzapine at therapeutic levels,
deep vein thrombosis.
Victoria Police
24, — The circumstances of Mr Finck’s death were subject to investigation by Victoria Policc.
A coronial brief of evidence was submitted to the Court in August 2008, containing statements from Mr Noel Finck, and from past and present employees of Eastern Health including Nurse Lankford, Dr M Hashim, Dr de Silva, Dr Gandhi, Dr Ambreem Memon, Dr Xiang Hua Loh, Dr Peter Sloan and Dr Vinod Aiyappan.
Clinical Liaison Service
25, The Clinical Liaison Service’ reviewed the circumstances of Mr Finck’s death on behalf of the Coroner in relation to the issue of whether it was reasonable and appropriate in the circumstances for Mr Finck to have been admitted to the Psychiatric ward rather than a
medical ward.
Nurse, as Nurse Lankford was,!!
available.
Issues identified for further exploration
Psychiatric ward with input from medical staff, and if the latter was appropriate, whether
10 Tho Clinical Liaison Service (CLS), now known as the [lealth and Medical Investigation Team (HMIT) is a unique initiative of the Coroners Court of Victoria and the Victorian Institute of Forensic Medicine (VIFM) to improve patient safety. HMIT is staffed by practising Physicians and Nurses who are independent of the health professionals or institutions involved. They assist the Coroner’s investigation of deaths occurring in a healthcare setting by evaluating the clinical management and care provided and identifying areas of improvement similar deaths may be avoided in the future.
Nurse Lankford gave evidence that there are only five patients in the HDU at any time, and two Nurses; transcript, p.
his medical management as a Psychiatric patient was adequate and appropriate in the
circumstances.
29, A Directions Hearing was held in March 2011 at which Interested Partics were informed of the issues identified by the investigation into the death of Mr Finck.
INQUEST Jurisdiction
From 1 November 2009, the Coroners Act 2008 (Vic) (the Act) has applicd to the finalisation of investigations into deaths that occurred prior to the new Act commencement.'* In the preamble to the Act, the role of the coronial system in Victoria is stated to involve the independent investigation of deaths for the purpose of finding the causes of those deaths and to contribute to the reduction of the number of preventable deaths and the promotion of public health and safety and the administration of justice.
Reference to preventable deaths and public health and safety are mentioned in other
sections of the Act.'*
relates to the context in which the death occurred.
Evidence at Inquest
c. Dr Piyumali de Silva, Psychiatric Registrar at Maroondah Hospital at the time of Mr Finck’s death;
d. Dr Peter Sloan, Director of Medical Services, Maroondah Hospital;
" Section 119 and Schedule 1 — Coroners Act 2008.
3 See for example, sections 67(3), 72 (1) and (2).
4 Section 67(1).
e. Dr Ashu Gandhi, Consultant Psychiatrist, Eastern Health; and
f. Dr Xiang Hua Loh, Endocrinology Resident at Maroondah Hospital at the time of Mr Finck’s death.
Concerns of Mr Finck’s family
Mr Noel Finck, on behalf of his family raised concerns regarding whether his brother’s apparent sedation and consequent immobilisation could have contributed to the
development of DVT.
Events prior to Mr Finck’s admission to Maroondah Hospital
37,
38,
Mr Noel Finck described his brother as a fairly fit man, despite his stocky appearance,
that used walking as his primary mode of transport.
Mr Noel Finck also described how his brother had spent several days in the sccurc area of the same Psychiatric ward approximately five ycars prior to his death. Mr Noel Finck described that this admission occurred in the context of excess alcohol consumption
following the break-up of his marriage.
Mr Noel Finck stated that his brother appeared happy living with his housemates, but noted that they had observed some unusual behaviour from his leading up to the night that he walked to his Aunt’s home. It is possible that Mr Finck ceased taking his
psychotropic medication around this time.
Mr Nocl Finck in his statement to the Court stated that his brother “struggled occasionally with mental health issues throughout his life but had been fine for 5 ycars
and many years before that occurrence.”
Various estimates were heard in respect of how far Mr Finck walked overnight, however it was clearly a significant distance, part of which he spent barcfoot for an uncertain
reason.
Medical care received while in Maroondah Hospital
Upon admission to Maroondah Hospital, Mr Finck was recommenced on his usual medication, Aripripazole 15mg daily. It was noted that he was rigid, especially in his
upper limbs. Medical records describe him as appearing perplexed, with poverty of
'S Statement of Mr Noel Finck dated 18 July 2008, coronial brief of evidence, p. 18.
40,
Al.
43,
44,
thoughts and blunted affect. Mr Noel Finck described him as almost catatonic.'® Ilis psychotic symptoms were listed as auditory hallucinations, diminished speech and facial expression, fearfulness, bizarre thoughts and behaviour, including unusual body posture,
slowing of movements, noting his episode of walking tens of kilometres barefoot.'7
In the morming of 18 September 2007, Mr Finck was reviewed by the Endocrinology House Medical Officer who requested a review by the medical Registrar, Dr Vinod Aiyappan, who reviewed him at 12.00pm. He noted that if Mr Finck’s urea and electrolytes results worsened or the CK levels rose, Mr Finck could be transferred to a medical ward after discussion with medical staff. Dr de Silva noted that Mr Finck’s
blood tests results proceeded to improve.'*
By the time that Mr Finck was transferred to the LDU on 19 September 2007, he was still requiring full nursing care and 15 minute visual observations.'? He had been placed
on a fluid balance chart.
When Nurse Lankford took Mr Finck’s observations at 4.30pm, she was concerned about tachycardia and hypertension.” She immediately asked Dr Harms to review Mr
Finck.
At the commencement of her shift, Nurse Lankford placed Mr Finck in a position within in his room where he was viewable from the Nurse’s station.”! Nurse Lankford gave evidence that as an enrolled Nurse, it was her responsibility to communicate any concerns regarding her patients to the unit doctor, and that it was “above her head” to
call in a physician from another unit, and not her responsibility to escalate situations.”
Dr de Silva was rostered on an “after-hours” shift that day, with her shift commencing at 5.30pm. According to her statement, she spent the first hour of her shift completing paperwork in her office. At approximately 6.30pm, she was approached by Dr Harms
and asked to review Mr Finck. Her evidence states that Dr Harms requested her to
© Transcript p. 9.
"7 Statement of Dr Piyumali de Silva, coronial brief of evidence, p. 13.
18 Transcript, p. 60.
© Transcript, p. 22, 41 and 44,
? Nurse Lankford was particularly concerned with the diastolic blood pressure reading of 120mmHg; transcript p.23
and 32,
2 ‘Vranscript, p. 31.
2 ‘Transcript, p. 36-38.
assess Mr Finck at approximately 7.30pm. He also advised her of Mr Finck’s room
number.
7,43 and 7.45pm.
admitted under psychiatric care. He stated;
“When a patient is to be admitted to hospital and has both medical and psychiatric diagnoses then a judgement is made between the psychiatry service and the medical unit as to the appropriate location of the patient in the hospital. The patient may be admitted to an acute medical bed with support from the psychiatry service or to a
psychiatry bed with support from the medical unit.”
Psychiatric ward and sit or lay in one place for an extended period.
responsibility “to manage both medical and psychiatric issues”.”°
assessment by duly qualified medical staff’.
3 Nurse Lankford stated that she had not taken other vital sign observations from 4.30pm until this time; transcript, p.25, and that Mr T'inck required four hourly vital signs observations; transcript, p. 28 and 29.
4 Transcript, p. 52-53.
3 Transcript, p. 60.
Sedation
Dr Gandhi was asked in evidence whether Mr Finck was sedated for the first few days upon his admission to the Psychiatric unit, or whether his apparent catatonic state was rather part of his presentation. Dr Gandhi explained that Mr Finck’s presentation was a result of a combination of factors, including him having travelled a long distance, a possible infection and the possibility that some of the medications provided in hospital may have affected his sedation level. Dr Gandhi stated that his presenting illness itself
possibly had features of a person who was sedated.”
Dr Gandhi gave evidence that some of the prescribed medication (diazepam, temazepam and olanzapine) was to treat Mr Finck’s agitation rather than to sedate him.”* Dr Gandhi was.also of the view that these medications were prescribed at relatively low doses.”” According to Dr Gandhi, the other medication, Aripiprazole (abilify), that Mr Finck was prescribed and had been taking for some time previously, was unlikely to result in
sedation.
Dr Gandhi stated that Mr Finck was observed to be responding to internal stimulation, which can sometimes misrepresent as patients being sedated.*” Dr Gandhi also explained that less severe catatonia, which he stated Mr Finck may have been experiencing, can
sometime be misinterpreted as psychotic symptoms or sedation.*!
Risk of DVT
Dr de Silva stated that she would be concerned about a DVT developing in a patient who is bedbound for more than 24 hours.’ She noted that although the risk of DVT raises when someone is dehydrated,? it was her impression that Mr Finck was mobilising, and
accordingly would not have considered that DVT prophylaxis was indicated.** Dr de
*6 Transcript, 27 Transcript, 8 Transcript, ® Transcript, »° Transcript, 3! Transcript, >» Transcript, 33 Transoripl,
p. 110.
p. 125.
p. 133-134.
p. 125, and 134.
p. 125.
p. 138.
p. 55-56,
p. 56.
p. 57,
Silva did not consider that it was clinically indicated to provide Mr Finck with intravenous fluid rehydration, as he was drinking reasonable volumes, and his blood test
results were improving.
Dr Sloan similarly stated that Mr Finck would not have been categorised as a patient with “significant immobility’, as he was toileting himself independently, therefore he considered the risk of immobility associated with DVT development would not apply to
Mtr Finck’s situation.*®
Dr de Silva conceded that DVT might be more difficult to diagnose in a Psychiatric setting where a patient is possibly not engaging sufficiently to communicate any relevant symptoms,’” however conceded that there would be no difference in his capacity to
discriminate between calf pain and epigastric pain.*®
Dr de Silva noted that it may have been challenging to nurse Mr Finck in a medical ward, due to his unpredictability, possible wandering behaviour, and the less frequent visual observations generally provided to medical unit patients.°? Dr Sloan however stated, that while he did not have a “great feel”? for Mr Finck’s mental health status, it was possible to have nursed him on the medical ward, that if (Psychiatric) patients are required to be on a medical ward, then “we provide them with additional nursing staff’, Dr Sloan however stated that there was nothing in Mr Finck’s presentation that
suggested he should have been treated in a medical ward.*!
Dr de Silva did not consider Mr Finck’s blood test resulis would indicate that a DVT was present,” nor did she consider there could be a relationship between his reported
epigastric pain and the DVT.“ Dr de Silva stated that there was no indication that Mr
® Transeri
3 Tyanseri factors.
37 Transcri 8 Transcri ® Transeri
*" Transcript,
” Transcript,
8 Transeri
pt,
pt,
pt, pt, pt,
pt,
pt,
p. 60.
. 101 and 116 where Dr Sloan also notes Mr Finck’s relatively young age in reference to known risk
a
p. 58 p. 68.
p. 58-59, 97.
97-98,
p. 68.
Page 12 of 17.
Finck was going to develop a DVT from the diagnostic results or observations.“* Dr
Sloan stated that tachycardia is not a symptom of DvT.*
embolism from DVT can occur without any warnin pA”
Dr Sloan explained that muscle breakdown, referred to as rhabdomyolysis, carries with it an opposite risk to clotting/DVT formation, rather the risk of disseminated intravascular
coagulation (DIC), a coagulation defect where patients bleed rather than clot.”
for DVT formation or provided with prophylaxis therapy.”
Psychiatric patients and DVT formation.” Incident Review
: : 51 review. For completeness, they include;
d, It is recommended that all Mental Health patients have a baseline set of
clinical observations and a physical assessment on arrival to the Emergency
“4 Transcript, p. 75.
** Transcript, p. 103. Dr Sloan went further and stated he considered the tachycardia was related to Mr Finck being thyrotoxic (p. 118).
“© Transcript, p. 75.
‘T Transcript, p. 97.
48 Transcript, p. 113-114.
® Transcript, p. 114.
°° Transcript, p. 128.
*! Although Ms Foy on behalf of Eastern Health initially canvassed an intention to make a submission pursuant to section 115(3) of the Act (transcript p. 4) and mentioned certain aspects of the report that highlighted Eastern Health’s intention that it remain an internal document and not be distributed (transcript p. 83-86), no formal application was made with respect to the document provided to the Court, during the Inquest or at the conclusion of the Inquest, when the document was specifically discussed.
Department, This is to be documented in the clients notes Emergency
Management orm MH/162.
Action: Dr Peter Archer Maroondah Hospital Director Emergency Medicine to provide information and education to all Emergency Medical staff as to the above requirements for Mental Health clients on admissions to the Maroondah Hospital
Emergency Department.
MH222 Chart.
Action: Dr Paul Katz Associate Professor astern Health Mental Health Service to
disseminate this information to all Mental Health clinicians in the Service.
Action: Mental Health Services Acute Operation’ Manager Mark Thornett to reinforce to all clinicians the importance of adherence to Kastern Health Policy for Clinical Risk Assessment and Review in relation to admission and management of all
inpatient clients.
Finck did not meet any of the accepted hospital guidelines for DVT prophylaxis.~°
lranscript, p. 94-95.
Transcript, p. 95.
Transcript, p. 95.
Transeript, p. 95-96.
Dr Gandhi stated that, based on discussions he has had with the Nurse Unit Manager of the Psychiatric unit, clinical risk assessments are completed upon admission, and clinical risk reviews upon the change of each shift (three times per day), whereas at the time of
Mr Finck’s death, the reviews were completed daily.*°
Submissions
67,
Oral submissions were received from Ms Deborah Foy of Counsel on behalf of Eastern Health on the final day of the Inquest. Ms Foy submitted that the medical care provided to Mr Finck was of an adequate standard and that he was appropriately followed up and reviewed by at least two medical practitioners every day during his admission.°’ Ms Foy submitted that a very thorough process of differential diagnoses was undertaken by all
medical practitioners with respect to Mr Finck’s symptoms.**
Ms Foy submitted that Mr Finck’s tragic clinical course was not diagnosable; there was no history which would have prompted hospital staff to consider that he had a DVT.” Ms Foy submitted that there is no evidence that suggests that moving Mr Finck to a medical ward would have been able to prevent his death, and that it is probable that he
reccived more nursing care in the HDU than he might have on a medical ward.”
Ms Foy therefore submitted that there should be no criticism directed to the medical or
nursing staff involved in Mr Finck’s care.”
While I accept that the medications prescribed to Mr Finck whilst an inpatient at the Psychiatric unit
can have a slight sedating effect,” I do not accept that Mr Finck was purposely sedated by staff at
Eastern Health and that any perceived sedation was morc likcly to be mild catatonic symptoms
linked to Mr Finck’s acute psychotic episode, I accept that any medication prescribed to him during
his admission, at relatively low doses, was provided to treat agitation rathcr then to promote
6 Transcript, p. 142-143.
"Transcript, p. 165.
» Transcript, p. 165.
© Transcript, p. 166.
*! Transcript, p. 166.
© Transcript, p. 134,
sedation. T also accept on the weight of the evidence that Mr Finck’s objective sedated appcarance
was likely an organic part of his presenting illness.
I am satisfied that Mr Finck’s clinical presentation prior to his death was not due to pulmonary embolism/decp vein thrombosis and that this condition could not have been predicted on the evidence available to medical staff at that time. I accordingly find that it was appropriate to treat Mr Finck in the West Ward Psychiatric unit with regular and appropriate monitoring by medical doctors. T also find that the medical management of Mr Finck, an involuntary Psychiatric patient,
was reasonable and appropriate in the circumstances.
Accordingly, I make no adverse findings against any Eastern Health staff involved in his care.
The investigation into Mr Finck’s death did not identify evidence that the outcome would have been
different if Mr Finck had been transferred to a medical ward.
I am satisfied that the recommendations made by and implemented at Eastern Health followed a proper review of the circumstances of Mr Finck’s death, and are, although not overwhelmingly
specific, restorative in nature.
J accept and adopt the medical cause of death as identified by Dr Sarah Parson and find that Paul Bernard Fink died from natural causes, being pulmonary thromboembolism and deep vein
thrombosis.
I direct that these Findings be published on the internet.
T direct that a copy of this finding be provided to the following: Mrs Loretta Finck
Mr Nocl Finck
Ms Sue Allen, Chicf Counsel, Eastern Health
Dr Paul Katz, Eastern Health
Ms Deborah Ellis of Counsel
Dr Mark Oakley Browne, Office of the Chief Psychiatrist
Police Coronial Support Unit Leading Senior Constable Allan Langley
Signature:
Date: 26 February 2015
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